| Causes |
The
direct cause is due to bubble formation and growth within body
organs and tissues. Mainly seen in divers but can be seen in
high altitude pilots or astronauts as well as persons working
in compressed air environments. The exact mechanisms are not
fully understood but rapid ascents from depth or sudden decompression
as well as incorrect decompression procedures do seem to be
a cause. Effects differ from person to person but varying predisposing
factors are thought to heavily contribute to DCI even with correct
procedures. Breath holding and air trapping in the lungs during
ascent or decompression may also lead to DCI as well as some
heart defects. |
Sources
|
Breathing
compressed gases under pressure |
Signs
& Symptoms |
Altered
consciousness, balance and coordination problems, memory problems,
headache, nausea and vomiting, vertigo, numbness and tingling,
joint pain, unusual fatigue, paralysis, skin rashes, difficulty
walking/standing, muscular weakness, difficulty urinating, difficulty
breathing |
HBO/recompression
in the case of decompression illness/ sickness |
Breathing
oxygen under pressure allows the inert gas bubbles to ‘offgas'
and be removed from the body much quicker. HBO will also reduce
swelling caused by bubble damage as well as oxygenate hypoxic
tissues. The pressure will also shrink the size of the bubbles.
|
Pre
hospital treatment |
Ensure
safe rescue, ABCs, High flow oxygen (preferably by demand valve),
oral fluids such as water or isotonic sports drinks, Gain I.V.
access (medical professional), I.V. fluids if safe (medical
professional) Pain killing drugs or analgesic gases should not
be given |
Referral
Criteria for Hyperbaric Oxygen Therapy/ Recompression
|
As
the symptoms for DCI are so diverse, any symptom that cannot
be readily explained after diving or other hyperbaric exposure
should be assumed to be DCI until proven otherwise. Any symptoms
that are suspected to have been caused by DCI should be treated
in a hyperbaric chamber. Many believe that DCI can only be treated
whilst in the acute phase (no longer than 7-10 days after symptom
onset) |
Duration
of
treatment |
Standard
treatment usually involves an initial session of Hyperbaric
Oxygen Therapy of between 2 and 7 hours duration at pressures
of between 2.8 and 4 ATA. Patients are then reassessed and further
treatment is given if necessary until all symptoms have resolved
or they reach a plateau (stay the same) |
Aim
of treatment |
Reversal
of symptoms and prevention of permanent damage tissues and organs,
reduction of possible long-term effects. |